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It is a 3-in-1 reference e-book. It provides a whole clinical dictionary overlaying 1000s of phrases and expressions on the subject of mind harm. It additionally provides broad lists of bibliographic citations. eventually, it presents details to clients on find out how to replace their wisdom utilizing a variety of net assets. The booklet is designed for physicians, clinical scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to familiarize yourself with learn devoted to mind harm. in the event that your time is efficacious, this booklet is for you. First, you won't waste time looking out the web whereas lacking loads of suitable info. moment, the e-book additionally saves you time indexing and defining entries. eventually, you won't waste money and time printing hundreds and hundreds of websites.
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Additional resources for Brain Damage - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
In addition, changes in BB transport characteristics may influence the transport of clinically useful medications used to either treat the ischemic episode or medicate other disease stages. Our major hypothesis is that hypoxia/aglycemia and reperfusion (associated with stroke) alters both the expression of BB specific proteins and the permeability and transport of critical solutes across the BBB, thereby contributing to the development and severity of stroke. To investigate this hypothesis, our major objective and specific aims are to investigate and quantify the effect of hypoxia/aglycemia/reoxygenation on the endothelial cells of the BBB by quantifying effects on cell resistance, permeability, tight junctional protein/cytoskeletal protein expression and transcriptional/posttranscriptional expression of specific proteins associated with hypoxia.
Generate_Screen • Project Title: FETAL BRAIN INJURY FROM OXIDANTS FOLLOWING ACUTE HYPOXIA Principal Investigator & Institution: Tan, Sidhartha; Evanston Hospital Evanston, Il 60201 Timing: Fiscal Year 2001; Project Start 01-JAN-1997; Project End 31-DEC-2002 Summary: Irreversible brain damage to the fetus frequently results from prolonged ischemia/hypoxia during pregnancy. Oxidants produced during ischemia- reperfusion can cause significant tissue damage if they overwhelm the body's antioxidant defenses.
It is widely believed that learning and memory are mediated by dynamic changes in the brain. Long-term potentiation (LTP) is an activity-dependent form of synaptic plasticity thought to be to be the most plausible mechanism for learning and memory. LTP was first discovered in the hippocampus, a neural structure associated with learning and memory. Interestingly, aging is accompanied by impairments in both hippocampal-dependent learning and LTP. Dietary restriction (DR) is the only environmental manipulation known to extend lifespan in all mammals studied.