Intraventricular Conduction Disturbances by Paolo Alboni M.D. (auth.)

By Paolo Alboni M.D. (auth.)

Rhythm was once the 1st expression of cardiac task which fell lower than man's obser­ vation, and the heartbeat has constantly represented the very essence of lifestyles itself because it hurries up or slows in the course of moments of leisure, attempt, pleasure and soreness till it involves a halt in the intervening time of demise. definitely the heartbeat used to be, for a substantial time, the single semiological aspect wherein guy may possibly interpret the dysfunctions of the organism. Even after hundreds of thousands of years, learn into the changes of cardiac rhythm nonetheless holds a definite fascination, even if glossy tools and complex units have replaced the method of such experiences. purely approximately ten years in the past, the in­ terpretation of arrhythmias was once acquired through a customary electrocardiogram to which experimental findings on animals have been nonetheless arbitrarily utilized. however, the success of diagnostic judgements used to be of little significance open air the sector of natural highbrow hypothesis in view that legitimate therapeutical remedies have been missing. at the moment, via electrophysiological investigations, you can actually be sure and money 'in situ' the various kinds and mechanisms in which rhythm and conduction problems are manifested. therefore, reaching this kind of technique that may be outlined as a 'functional biopsy', we're allowed to place ahead extra actual healing symptoms now to be had utilizing either pharmacological and electric treatment.

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Extra info for Intraventricular Conduction Disturbances

Sample text

The wave fronts then advance into the right ventricle and reach the anterior wall and the pulmonary conus. The 31 isochrones are more widely spaced on the posterior side of the right ventricle than anteriorly, indicating a more rapid spread in the posterior wall [9]. ELECTROCARDIOGRAPHIC FEATURES The following ECG changes are observed in the presence of RBBB: - QRS interval prolongation to 120 msec or more. - Electrical axis between 0° and 90°. - rSR, rsR, R, Rs or Rr morphology, with terminal slurring in right precordial leads.

The variations of the LBBB do not appear rate-dependent. The QRS complexes conducted with incomplete LBBB measure 100 msec; in leads I, aVF, Vs and V6 an initial slurring and a terminal one are present; Q waves are not evident normal time lag of right septal activation after onset of excitation on the left septal surface therefore tends to be abolished, with the result that the initial forces due to left-to-right septal depolarization are opposed and, in part at least, neutralized. This circumstance is responsible for the disappearance of the Q wave in V6 lead.

Left ventricular hypertrophy in the presence of right bundle branch block. Circulation 56: 391, 1977 73. Scott RC: Left bundle branch block. A clinical assesment. Am Heart J 70: 535, 1965 74. Abildskow JA, Jackson CE, Burch GE, Cronvich JA: The spatial vectorcardiogram in right bundle branch block. Circulation 31: 600, 1951 75. Grishman A, Scherlis L: Spatial vectorcardiography. Philadelphia: WB Saunders, 1952 76. Massie A, Walsh TJ: Clinical vectorcardiography. Chicago: Year Book Medical Publishers, 1960 77.

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