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Neuropsychopharmacology: A Tribute to Joseph T. Coyle by Robert Schwarcz

By Robert Schwarcz

Neuropsychopharmacology: A Tribute to Joseph T. Coyle is a brand new quantity from Advances in Pharmacology providing studies of contemporary breakthroughs in glutamate pharmacology and a tribute to at least one of the main influential neuroscientists of our occasions. With various chapters and the easiest authors within the box, the amount is an important source for pharmacologists, immunologists, and biochemists alike.

  • Features contributions from the easiest authors within the field
  • Provides a necessary source for pharmacologists, immunologists, and biochemists
  • Includes new ways for diagnosing and treating significant neurological and psychiatric diseases
  • Features a tribute to at least one of the main influential neuroscientists of our times

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Neuropsychopharmacology: A Tribute to Joseph T. Coyle

Neuropsychopharmacology: A Tribute to Joseph T. Coyle is a brand new quantity from Advances in Pharmacology proposing stories of modern breakthroughs in glutamate pharmacology and a tribute to at least one of the main influential neuroscientists of our instances. With quite a few chapters and the simplest authors within the box, the quantity is a vital source for pharmacologists, immunologists, and biochemists alike.

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This approach was further supported by evidence suggesting that clozapine’s unique therapeutic efficacy might in part result from effects on glutamatergic transmission. , 2008); this model predicted that reduction of glutamate release by lamotrigine or mGlu2/3 agonists might also produce therapeutic benefit. , 2014). More recently, genetic studies in large samples of individuals with schizophrenia have implicated a diverse array of more than 100 SNPS associated with neurodevelopment, synaptic plasticity, and inflammation, many of which are shared in common with other neurodevelopmental psychiatric disorders (Hall, Trent, Thomas, O’Donovan, & Owen, 2015; Schizophrenia Working Group of the Psychiatric Genomics Consortium, 2014), while postmortem studies consistently identify deficits in inhibitory transmission via GABAergic interneurons (Lewis, 2009).

Bruno, J. P. (2012). Acute elevations of brain kynurenic acid impair cognitive flexibility: Normalization by the alpha7 positive modulator galantamine. Psychopharmacology, 220, 627–637. , Pereira, E. , Arruda, E. , Almeida, L. , et al. (2004). Targeted deletion of the kynurenine aminotransferase ii gene reveals a critical role of endogenous kynurenic acid in the regulation of synaptic transmission via alpha7 nicotinic receptors in the hippocampus. Journal of Neuroscience, 24, 4635–4648. , & Schwarcz, R.

2014). Role of glutamate transporters in redox homeostasis of the brain. Neurochemistry International, 73, 181–191. , & Mika, J. (2016). Pharmacological kynurenine 3-monooxygenase enzyme inhibition significantly reduces neuropathic pain in a rat model. Neuropharmacology, 102, 80–91. Rothstein, J. , Pardo, C. , Bristol, L. , & Kuncl, R. W. (1996). Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate. Neuron, 16, 675–686. , Cesura, A.

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